In this paper, a Markov model of finite fuzzy neuronal assembly activity is presented, and Poisson approximation is considered.
The major lesions included dissolution of Nissl bodies, pyknosis, and incrustation of neuronal surface in the cerebral cortex, thalamus, hippocampus, cerebellum and spinal cord.
Study Design. An analysis of pain behavior and neuronal apoptosis in the dorsal root ganglion (DRG) following crush injury to the L5 nerve root, with or without surgical sympathectomy.
Most scientists have viewed electrical fields within the brain as the simple byproducts of neuronal activity.
The precise anatomic location of neuronal injury and the etiology of the clinical manifestation were poorly understood.
Endogenous and exogenous opioids can influence and modulate neuronal and glial cell function via an opioid receptor mediated mechanism, leading to either protection or damage of the brain.
Any search for neuronal replication will have to take place at a different level - perhaps in the patterns of connections between groups of neurons or in their patterns of activity.
The mechanism of hypsarrhythmia EEG resulted from brainstem dysfunction on the neuronal population level was studied.
Because the existing of neuronal plasticity, the majority of dysphagia after cerebral infarction may recover by itself, but dysphagia will ultimately affect the prognosis of patients.
Conclusion CACNA1H gene G773D mutation is able to increase calcium current and neuronal excitability.
These results indicate that VPA induces a specific differentiation of C6 cells toward a neuronal-like phenotype.
The effect of nerve growth factor on neuronal apoptosis and the expression of P75NTR and TrkA of neuronal cell which is induced by oxyhemoglobin in mice
Topics include: neural induction and pattern formation, cell lineage and fate determination, neuronal migration, axon guidance, synapse formation and stabilization, activity-dependent development and critical periods, development of behavior.
BACKGROUND: Cerebral ischemia and reperfusion affects not only cellular necrosis at acute stage, but also delayed neuronal apoptosis in central neural system.
MAIN OUTCOME MEASURES① Changes of neuronal cytoplasmic free calcium concentration.
Objective To investigate the proliferation and differentiation of neuronal progenitor cells in the rat brain after cerebral ischemia.
Many ISDN members are also interested in aspects of aging, as well as neuronal dysfunctions associated with trauma or disease.
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