Objective: To investigate the effect of inhaled nitric oxide (NO) on pulmonary edema in dogs after smoke inhalation injury.
Objective To explore the role and significance of apoptosis, pro-apoptotic genes and apoptosis suppressor genes in the pathogenesis and development of inhalation injury.
Objective to evaluate the effects of inhalation injury patients treated with respiratory function training.
The establishment of the method to culture pulmonary microvascular endothelial cells (PMVECs) is important for the study of inhalation injury.
All the rats except those in pre-injury group were subjected to smoke inhalation injury.
To explore the role of apoptosis, apoptosis regulating genes in the pathogenesis and development of smoke inhalation injury.
Method: The data of 110 patients with inhalation injury from 1992 to 2004 were retrospectively analyzed.
Methods: ct scans of the lung were performed on 16 healthy mongrel dogs before and after steam inhalation injury and lung tissue sections were prepared for histopathologic examination.
Conclusion Early and prophylactic tracheostomy should be advocated in patients suspected to be with moderate or severe inhalation injury.
Nurses of burning department must pay attention to the respiratory function training for patients with inhalation injury so as to improve therapeutic effect and enhance self-efficacy.
Xenon lung scans may be helpful in the early diagnosis of inhalation injury.
Objective To investigate the protective effects of insulin on smoke inhalation injury in rats.
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