研究者追蹤185位有CLL表現型MBL與淋巴球數目增生的病患,且監測他們平均達6.7年;
結論:這些結果首次提示MBL在動脈粥樣硬化早期局域*大量表達。
However, the relationship between structure and functions of MBL and the mechanisms by which MBL structure gene mutations cause opsonic deficiency are to be clarified.
Interaction of Mannan-binding Lectin with MBL-associated Serine Proteases;
The MBL has an infrared countermeasures capability that provides laser jamming in all three key threat bands.
MBL具有一種紅外干擾能力,可以在所有的三個關鍵威脅波段中提供激光干擾。
目前,MBL的結構-功能關係未明,MBL基因突變引起調理吞噬缺損的機制不清,這些均需進一步研究予以闡明。