Study on the Mechanisms of α-synuclein Aggregation in Rotenone Induced Parkinson s Disease;
These include inflammatory stimulation by interaction of alpha-synuclein with microglia and astrocytes and a suppressive action by nonsteroidal anti-inflammatory drugs on dopamine quinone formation.
Establishment of Tetracycline-controlled A30P Mutation α-synuclein Transgenic Mice
Mechanism of curcumin's protection of dopaminergic cells from rotenone-induced injury by inhibiting α-synuclein aggregation
Changes of α-synuclein After Accumulation
Interaction of DJ-1 with Small peptide to Inhibit the Aggregation of Alpha-synuclein in
Manganese alters α-synuclein expression in rat brain
Experimental Study on the Effects and Possible Mechanism of Alpha-synuclein to MnCl_2-treated PC12 Cells in Vitro;
Dual effects of different concentrations of α-synuclein on the neurotoxicity of 6-hydroxydopamine in SH-SY5Y cells
It is thought that clumps of alpha-synuclein accumulate because brain cells cannot remove them through autophagy - a type of programmed cell death.
Construction of the eukaryotic expression vector of pEGFP-N1-α-synuclein
Transcriptional Regulation of Tyrosinase by α-synuclein and Inhibitors