Effects of contrast media and mannitol on rat glomerular endothelial cells
Valsartan inhibits the expression of connective tissue growth factor in rat glomerular mesangial cells incubated with high concentration glucose
Inhibition of fenofibrate or rosiglitazone on production of extracellular matrix and oxidative stress of glomerular mesangial cells incubated with high concentration of glucose
They are glomerular nephropathy with the disease, peripheral and central nervous system lesions, skin rashes, Raynaud's phenomenon of the pathological basis.
The glomerular filtration rate remains relatively constant.
Correlation between glomerular filtration rate and multiple risk factors in newly diagnosed type 2 diabetic patients
Effects of dexamethasone on changes of monolayer permeability and F-actin of glomerular endothelial cells induced by Angiotensin Ⅱ
The pathological changes were characterized by formation of antigen-antibody complexes both in the alveoli basement-membrane and the glomerular basement-membrane.
tubulo-glomerular feedback
Significance of renal interstitial foam cells in patients with glomerular disease
Effect of ubiquitin-proteasome pathway on expression of Smad7 in cultured glomerular mesangial cells induced by high concentration of glucose
The results presented that the damage of glomerular filtration membrane had already taken place and, that the epithelial cells of the proximal tubule showed cloudy swelling at 1 week.
Relationship between glomerular nephrin expression and oxidative stress reaction in rats with adriamycin-induced nephrosis
[Re-sults] 27 children with CRF were diagnosed as acquired glomerular diseases,including 14 cases of nephritic syndrome and 9 cases of chronic glomerulonephritis.
Immune response is one of the main causes of glomerular diseases. Immunosuppressive agents are an important means to treat glomerular diseases.
The histopathological changes of the kidneys included renal tubular necrosis and detachment, formation of casts, glomerular ischemia and shrinkage and infiltration of large amount of PMN in LPS group.
Effect of bufalin on expressions of IL-6 and TGF-β1 in glomerular mesangial cells of rats
When the glomerular/glou'merjulo/ destruction is extensive, serious impairment of renal/'ri:nl/ function results.
Conclusion PDGF may participate in glomerular cells proliferation and inflammatory cells infiltration in LN, and may be on factor in the different pathological type formation of LN.
Variation of nitrogen monoxidum concentration in patients with glomerular diseases
Clinical investigation of familial focal segmental glomerular sclerosis
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