“GABA”簡單造句，GABA造句子

Effects of 3,4-Dichlorophenyl-propenoyl-sec-butylamine on uptake and release of GABA

谷氨*脱羧酶(GAD)是合成GABA的主要限速酶。

結果表明：中縫背核內存在VIP樣、GABA樣免疫反應陽*的觸液神經元；

他和他的同事們已經跟蹤了氨基丁*（GABA）這一種方法，減少人們在恐慌症中出現的GABA可以有效的抑制恐慌症狀。

免疫電鏡發現在外毛細胞底部有GABA - IR陽*的傳出神經末梢及**的傳入、傳出神經末梢。

本試驗結果提示GABA能夠短期改善仔豬的採食量和日增重，並調節與採食關係較為密切的胃泌素變化。

In the retina of RCS rats, GABA-like immunoreactivity was observed in the inner plexiform layer, few GABA-ergic amacrine cells and the ganglion cell layer.

繼而，左旋巴**抑制場電位作用強於GABA。

讓對照組仔雞飲用蒸餾水，讓試驗組飲用0 0 5 %的氨酪*（GABA）蒸餾水。

異**一類的*物能夠作用於這類接收GABA的神經末梢，通過模擬和增強GABA引起的效果，起到抑制細胞活*的作用。

免疫細胞化學方法觀察了GABAA受體(gabaar)亞單位、GABA和GAD在大鼠三叉神經尾測亞核內的分佈及其匹配關係。

Glutamic acid decarboxylase (GAD) is a major GABA synthesizing enzyme.

Gabapentin is a new anti-epileptic agent whose chemical structure resembles that of GABA.

GABA nervous disorders, antrum-pylorus deficiencies, malnutrition factors, coincide mouth edema, when anti flow is the main pathological physiological and anatomical pathology foundation.

GABA陽*纖維主要集中於前外區、前背區背側部、前腹區梭形核和紋旁核及後部背側區域。

男人們永遠不能説出來但卻是想説的話：“你是不是有點經前綜合徵？” 經前綜合徵（PMS）的發生是由於體內偏重於情緒的荷爾蒙分泌過多，從而使大腦神經錯位造成的。血清素的平衡發生了變化。低水平的血清素與抑鬱有關；γ-氨基丁*（GABA)與焦慮有關；內啡肽是改變痛閥的影響神經系統的化學物質。簡言之，情感都是搖擺不定的，思維往往不注意事實。如果額葉無法適應新的情緒狀態，那麼它可以使人的行為進入無法預料的暴怒狀態。

It was found that the antagonist picrotoxin was unable to block the liver fluke to the excitatory effect of GABA, while it could partially block the roundworm to the inhibitory effect of GABA.

參加瑜珈鍛鍊的一組在研究結束後，GABA水平有了顯著提高。

RCS鼠則分佈於視網膜內叢狀層的少量神經纖維、極少量GABA陽*免疫反應的無長突細胞和節細胞層的部分纖維。

Participants in the yoga group had significantly increased levels of GABA at the end of the study.

本研究結果提示，胃中可能存在一種GABA攝取系統的負反饋式的自身調節機制。

The results indicated that GABA improved feed intake and average daily gain in a shortterm mode, which was involved in changed gastrin production modulated by GABA.

撤*反應的出現可能與*物對中樞神經系統中血清素、多巴*、去*腎上腺素、GABA、膽鹼能類遞質系統的影響有關。

Under TEM, GABA-IR positive efferent endings, negative efferent endings and negative afferent endings could be seen on the OHC bases.

The results suggest that possibly there is a mechanism of negative feedback autoregulation of GABA uptake system in mouse stomach.

它會釋放過量的伽馬氨基丁*(GABA)，以降低中風對受損位置神經元的刺激。

It releases extra amounts of GABA, which reduces the firing of neurons.

Drugs like propofol act on these GABA a receptors, mimicking and enhancing the effects of GABA, which inhibits cellular activity.